Download PDF by George F. Vande Woude (ed.), George Klein (ed.): Advances in Cancer Research, Vol. 61

By George F. Vande Woude (ed.), George Klein (ed.)

ISBN-10: 0120066610

ISBN-13: 9780120066612

Quantity sixty one of "Advances in melanoma study" contains insurance of the next: molecular ways to melanoma remedy, radiation resistance, melanoma prevention examine trials, mammalian gene amplification, retinoblastoma gene functionality, molecular genetic foundation of breast melanoma, and tumour advertising through okadaic acid type compounds.

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Stolley, P. , and Shapiro, S. J. Natl. Cancer Imt. 83, 355-358. ,and Ron, E. (1989). Gut 30, 650-655. , Freedman, L. , Schiffman, M. H . , e t a l . J. Natl. CancerImf. 82,17461752. , Hamilton, S. , Kinzler, K. , and Vogelstein, B. (1992). Science 256, 102-105. Sinkeldam, E. , Kuper, C. , Bosland, M. , Hollanders, V. M. , and Vedder, D. M. ( 1990). Cancer Res. 50, 1092- 1096. , Sorenson, A. , and Ford, M. H. (1988). A m . ] . Epidemiol. 128, 504-514. Sporn, M. B. (1976). Cancer Res. 36, 2699.

Mutant forms of p53 protein have an increased half-life. , 1990). An example of such staining is shown in Fig. 3. , 1991). Whether the association of immunohistochemically detectable p53 staining with the presence of a pointmutated p53 gene is absolute remains to be established. T h e percentage of breast carcinomas positive for p53 nuclear staining is 50-60%. , 1991; P. Devilee and C. Cornelisse, personal communication). In view of these figures, increased levels of p53 protein may also be the result of mechanisms other than p53 gene point mutations.

1990). An example of such staining is shown in Fig. 3. , 1991). Whether the association of immunohistochemically detectable p53 staining with the presence of a pointmutated p53 gene is absolute remains to be established. T h e percentage of breast carcinomas positive for p53 nuclear staining is 50-60%. , 1991; P. Devilee and C. Cornelisse, personal communication). In view of these figures, increased levels of p53 protein may also be the result of mechanisms other than p53 gene point mutations.

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Advances in Cancer Research, Vol. 61 by George F. Vande Woude (ed.), George Klein (ed.)


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